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regulation of cellular redox status via interaction with peroxiredoxin 6

Next, the steroid-receptor complex moves into the nucleus where it activates anti-inflammatory genes and represses pro-inflammatory genes by down-regulating their expression or inhibiting their transcription factors like NF-B and AP-1 (Figure 4)

In the nineties, a guinea pig model of emphysema was developed by means of chronic exposure to cigarette smoke [41]

490 Excessive inhibition of Nrf2 may not improve DOX-induced cardiomyopathy due to insufficient promotion of antioxidant gene expression, while its overactivation increases the breakdown of heme and ferrous iron levels in the HO-1-catalyzed manner, promoting ferroptosis

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